LncRNA MEG9 Promotes Inflammation and Liver Fibrosis Through S100A9 in Biliary Atresia

In brief

This research investigates the molecular mechanisms underlying biliary atresia, focusing on how long noncoding RNA MEG9 contributes to inflammation and liver fibrosis through S100A9 protein interactions. The study advances understanding of BA pathogenesis and may inform future therapeutic targets for this pediatric cholestatic liver disease.

Written by the GCMD Library team from the article.